Resumo

CARVALLO, Pamela et al. NAFLD and High Fructose Intake: A Review of Literature. Int. J. Morphol. [online]. 2017, vol.35, n.2, pp.676-683. ISSN 0717-9502.  http://dx.doi.org/10.4067/S0717-95022017000200047.

One of the most commonly used sweeteners is fructose. Fructose is directly metabolized in the liver and can be converted into glucose, later stored as glycogen constituting a source of energy for the hepatocytes. All excess fructose is converted into lipids by exerting a toxic effect on the liver, similar to that produced by excess of alcohol, and can cause nonalcoholic fatty liver (NAFLD). The aim of this review is to gather recent findings regarding the effect of fructose intake at high concentrations and its relationship with NAFLD.

Palavras-chave : Non-alcoholic fatty liver; Nonalcoholic steatohepatitis; Hepatic fibrosis; Fructose.

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